The hemodynamic mechanisms of high blood pressure are usually restricted to the learn of 3 dominant parameters: blood strain, cardiac output and vascular resis­ tance. for that reason, the improvement of high blood pressure is mostly analyzed when it comes to a 'struggle' among cardiac output and vascular resistance, leading to the classical development of standard cardiac output and elevated vascular resistance, hence indicating a discount within the quality of small arteries. besides the fact that, in the past years, the medical administration of high blood pressure has mostly converted those easy perspectives. whereas an sufficient keep an eye on of blood strain should be acquired with antihypertensive medicines, arterial problems might ensue, concerning in most cases the coronary stream and suggesting that numerous components of the cardiovascular approach are altered in high blood pressure. certainly, disturbances within the arterial and the venous method had already been spotted in animal high blood pressure. the elemental assumption during this booklet is that the final cardiovascular method is keen on the mechanisms of the increased blood strain in sufferers with high blood pressure: not just the guts and small arteries, but additionally the massive arteries and the venous procedure. hence, the subsequent issues are emphasised. First, the cardiovascular procedure in high blood pressure has to be studied not just by way of regular circulation but in addition by way of considering the pulsatile elements of the center and the arterial platforms. moment, arterial and venous compliances are altered in high blood pressure and possibly mirror intrinsic adjustments of the vascular wall.